Bilateral Internuclear Ophthalmoplegia Observed in Anti–GQ1b Antibody Syndrome

The anti–GQ1b antibody is responsible for Miller Fisher syndrome, which is characterized by the clinical triad of ataxia, ophthalmoplegia, and areflexia. The clinical spectrum of anti–GQ1b antibody syndrome continues to expand to include Bickerstaff brainstem encephalitis, Guillain-Barré syndrome with ophthalmoplegia, acute ophthalmoplegia without ataxia, acute vestibular syndrome, optic neuropathy, and acute sensory ataxic neuropathy in addition to the classic Miller Fisher syndrome. Patients with anti–GQ1b antibody syndrome may show ophthalmoplegia, such as internuclear ophthalmoplegia, from involvement of the central nervous system.

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Видео Bilateral Internuclear Ophthalmoplegia Observed in Anti–GQ1b Antibody Syndrome канала JAMA Network